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As mentioned previously, theseries of events that leads to the reprogramming of Tregs into TH17 T cells involves thesynergistic activities if IL-6 and TGF-?on degrading FOXP3 proteins thus inhibits its abilityto bind to the RORgammat gene and induce the TH17 phenotype.

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Rh30 polypeptideis a component of a larger (90 kDa) erythrocyte integralmembrane protein complex that includes Rh50 glyco-protein. One of the major questions is why does this CD4 T cell response failin response to late stage melanomas that are still producing these MHC-class II antigencomplexes on their cell surfaces? This is a complex question, but some of this failure has todo with the development of alternate MHC-class II antigen peptide processing pathways thatare developed by melanoma cells as the continue down the tumorgenesis pathway to late-stage disease.

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